In Alzheimer’s disease, glutamate excitotoxicity which can damage neurons through chronic activation is exacerbated by deficiency of ABCA7, the second strongest genetic risk factor for the disease. The findings highlight ABCA7 and glutamate–GABA signaling as potential therapeutic targets.
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Kevin PeacheyCost of living correspondent
В России ответили на имитирующие высадку на Украине учения НАТО18:04